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541
<i>Mertk</i><sup><i>-/-</i></sup> macrophages exhibit unpolarized distribution of filamentous actin and myosin II, and decreased FAK phosphorylation.
Published 2015“…Scale bars in (A) and (B), 5 μm. (C) Western blotting shows decreased FAK phosphorylation at tyrosine 397 after addition of apoptotic T cells for the indicated time points. …”
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542
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543
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544
Decreases in time from AHP to threshold account for increased firing of cPLA<sub>2</sub>α<sup>-/-</sup> SCG neurons.
Published 2018“…Both cPLA<sub>2</sub>α<sup>+/+</sup> (***<i>p</i> ≤ 6 x 10<sup>−5</sup>; n = 18) and cPLA<sub>2</sub>α<sup>-/-</sup> (**<i>p</i> ≤ 0.005; n = 8) neurons exhibit increases in firing frequency following Oxo-M. …”
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545
A Point Mutation in a lincRNA Upstream of <i>GDNF</i> Is Associated to a Canine Insensitivity to Pain: A Spontaneous Model for Human Sensory Neuropathies
Published 2016“…Clinical signs appear in young puppies and consist of acral analgesia, with or without sudden intense licking, biting and severe self-mutilation of the feet, whereas proprioception, motor abilities and spinal reflexes remain intact. Through a Genome Wide Association Study (GWAS) with 24 affected and 30 unaffected sporting dogs using the Canine HD 170K SNP array (Illumina), we identified a 1.8 Mb homozygous locus on canine chromosome 4 (adj. p-val = 2.5x10<sup>-6</sup>). …”
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546
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547
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548
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549
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550
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551
All data points from Fig 2.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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552
All data points from Fig 8.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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553
All data points from Fig 3.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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554
All data points from Fig 1.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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555
All data points from Fig 4.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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556
All data points from Fig 9.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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557
All data points from Fig 7.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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558
All data points from Fig 6.
Published 2025“…Following a remyelination period of 5 weeks, PV interneuron properties were only partially recovered, suggesting that transient juvenile demyelination leads to long-lasting impairments of PV interneuron function. …”
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559
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560