NTM infection induces mitochondrial damage and reduces mitochondria number. حسب Joel R. Frandsen (20685133)

الموضوعات:

Excel raw data. حسب Michael Getie (12400923)

"…The effectiveness of currently available antimicrobial is decreasing due to the increasing prevalence of resistant strains among bacterial isolates. …"

Uronic acid analysis-based screen identified IFA as an effective capsule inhibitor. حسب Tingting Wang (123983)

الموضوعات:

IFA did not affect LPS-induced inflammation response. حسب Tingting Wang (123983)

الموضوعات:

IFA abrogated capsule-associated hypermucoviscosity phenotypes of hypervirulent <i>K</i>. <i>pneumoniae</i>. حسب Tingting Wang (123983)

الموضوعات:

IFA effectively reduced the production of <i>K</i>. <i>pneumoniae</i> capsule. حسب Tingting Wang (123983)

الموضوعات:

IFA sensitized hypervirulent <i>K</i>. <i>pneumoniae</i> to complement-mediated killing by inhibiting capsule. حسب Tingting Wang (123983)

الموضوعات:

IFA disarmed capsule-mediated immune evasion. حسب Tingting Wang (123983)

الموضوعات:

Validation of distinct contribution of capsule and hypercapsule-associated HMV in IFA-conferred protection against hvKP infection using a <i>ΔrmpD</i>-K7 mutant. حسب Tingting Wang (123983)

الموضوعات:

IFA reduced the virulence of hypervirulent <i>K</i>. <i>pneumoniae</i> in <i>G</i>. <i>mellonella</i> larvae. حسب Tingting Wang (123983)

الموضوعات:

IFA treatment facilitated host immune clearance of hypervirulent <i>K</i>. <i>pneumoniae</i> by targeting capsule. حسب Tingting Wang (123983)

الموضوعات:

Single-phage therapy succeeds against phage resistance with a strong enough immune system. حسب Jacopo Marchi (7252499)

Image 6_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Image 5_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Image 1_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Image 4_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Image 7_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Data Sheet 1_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.docx حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Image 2_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.jpeg حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"

Data Sheet 2_Tumor-associated bacteria activate PRDX1-driven glycolysis to promote immune evasion and PD-1 antibody resistance in hepatocellular carcinoma.docx حسب Heng Zhang (320479)

"…Background<p>Recent studies have highlighted the presence of intratumoral bacteria in hepatocellular carcinoma (HCC), yet their contribution to immunotherapy resistance remains largely unexplored. This study investigates the mechanisms by which bacterial infection reshapes tumor metabolism to undermine the efficacy of anti-PD-1 therapy.…"