Data Distribution and Sample Clustering. af Jun Li (6494)

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Expression of CRGs in H9C2 DCM model. af Jun Li (6494)

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Validation of hub genes in dataset GSE5606. af Jun Li (6494)

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Flow chart of methodologies applied in the study. af Jun Li (6494)

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The expression profiles of 7 hub genes in GSE5606. af Jun Li (6494)

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GO and KEGG enrichment analysis of DECRGs. af Jun Li (6494)

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PPI network and identification of hub genes. af Jun Li (6494)

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Heatmap for the DECRGs. af Jun Li (6494)

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KEGG enrichment analysis of the DEGs in GSE267352. af Jun Li (6494)

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Colorimetric Response of Dithizone Product and Hexadecyl Trimethyl Ammonium Bromide Modified Gold Nanoparticle Dispersion to 10 Types of Heavy Metal Ions: Understanding the Involve... af Yumin Leng (1943116)

Biplot diagram from a partial redundancy analysis (RDA) summarizing the variation in diatom species composition explained by the host organism (macroalga or microfauna), after remo... af Roksana Majewska (756294)

Sapphormation workshop consent form af Niamh Moore (6855716)

“...<p>The Reanimating Data Project (2018-2020) involved a series of experiments in working with extracts from interviews with young women from 1989-1990. ...”

Age, gender, clinical BU lesion, and lesion location. af Carlos Capela (793868)

Improving upon mutual information by removing column bias. af Christopher A. Brown (248361)

“...The normalization reduces both the correlation between the product of the mean column mutual information (scatter plot, upper inset) and the product of the standard deviation of column mutual information (scatter plot, lower inset), but doesn't remove it entirely. <b>C.</b> ZNMI approximates the column normalized MI distributions (solid red line and solid blue line) as Gaussian distributions (dashed red line and dashed blue line), calculates a closed-form expression for the product of the two distributions (solid green line: kernel density estimate of product), and then z-scores the normalized mutual information (black solid vertical line) based on the Gaussian approximation of the product (dashed green line)....”

Schematic representation of glycosylation forms. af Yuliet Mazola (189934)

On modular forms and Domb numbers af J. M. Campbell (2181792)

“...These experimental results from Chan and Verrill involve the sequence of Domb numbers and were discovered via the PARI software. ...”

Comparison of predicted models of human NOX1 and experimentally resolved models of csNOX5, human DUOX1 and core human NOX2 transmembrane region. af Yezhou Liu (6718037)

(A) Schematic view of the chemical and enzymatic reactions involved in the TryR assay resulting in the reduction of the Elmman's reagent (DTNB) to the yellow-colored 2-nitro-5-thiobenzoic acid (TNB); (B) Same as above, showing the interference of cochlioquinone A resulting in a false positive readout due to its reaction with the reduced form of trypanothione, interrupting the chemical reduction of DTNB. af Fernanda Fraga Campos (268436)

“...<p>(A) Schematic view of the chemical and enzymatic reactions involved in the TryR assay resulting in the reduction of the Elmman's reagent (DTNB) to the yellow-colored 2-nitro-5-thiobenzoic acid (TNB); (B) Same as above, showing the interference of cochlioquinone A resulting in a false positive readout due to its reaction with the reduced form of trypanothione, interrupting the chemical reduction of DTNB....”

Channel-Forming Activities in the Glycosomal Fraction from the Bloodstream Form of <em>Trypanosoma brucei</em> af Melisa Gualdron-López (172625)

“...</p> <h3>Methods/Principal Findings</h3><p>We hypothesized that glycosomal membrane, similarly to membranes of yeast and mammalian peroxisomes, contains channel-forming proteins involved in the selective transfer of metabolites. ...”

Data_Sheet_1_CircRNA-ceRNA Network Revealing the Potential Regulatory Roles of CircRNA in Alzheimer’s Disease Involved the cGMP-PKG Signal Pathway.DOCX af Yuan Zhang (41832)

“...The characteristic pathologies include extracellular senile plaques formed by β-amyloid protein deposition, neurofibrillary tangles formed by hyperphosphorylation of tau protein, and neuronal loss with glial cell hyperplasia. ...”