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Visas 1 981 - 2 000 av 28 093 resultat för sökning 'form is (((((resolvent. OR involves.) OR involves.) OR resolveddss.) OR involved.) OR resolve.)', Sökningstid: 0,52s Förfina resultatet
  1. 1981
    Cells (GFP-Rab11a WT<sup>low</sup>) were mock-infected with PR8 virus for 12 h at an MOI of 3.

    Cells (GFP-Rab11a WT<sup>low</sup>) were mock-infected with PR8 virus for 12 h at an MOI of 3. av Sílvia Vale-Costa (282115)

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  2. 1982
    ATG9A is determinant for the correct shape of IAV inclusions.

    ATG9A is determinant for the correct shape of IAV inclusions. av Sílvia Vale-Costa (282115)

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  3. 1983
    ATG9A depletion arrests viral inclusions on microtubules.

    ATG9A depletion arrests viral inclusions on microtubules. av Sílvia Vale-Costa (282115)

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  4. 1984
    Viral inclusions are biomolecular condensates containing single- and double-membranes inside.

    Viral inclusions are biomolecular condensates containing single- and double-membranes inside. av Sílvia Vale-Costa (282115)

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  5. 1985
    ATG9A depletion affects viral genome assembly but not viral genome packaging.

    ATG9A depletion affects viral genome assembly but not viral genome packaging. av Sílvia Vale-Costa (282115)

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  6. 1986
    Cells (GFP-Rab11a WT<sup>low</sup>) were infected with PR8 virus for 12 h at an MOI of 3.

    Cells (GFP-Rab11a WT<sup>low</sup>) were infected with PR8 virus for 12 h at an MOI of 3. av Sílvia Vale-Costa (282115)

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  7. 1987
    ATG9A impacts viral inclusions independently of Rab11a–vRNP interaction.

    ATG9A impacts viral inclusions independently of Rab11a–vRNP interaction. av Sílvia Vale-Costa (282115)

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  8. 1988
    Cells (GFP-Rab11a WT<sup>low</sup>) were infected with PR8 virus for 12 h at an MOI of 3.

    Cells (GFP-Rab11a WT<sup>low</sup>) were infected with PR8 virus for 12 h at an MOI of 3. av Sílvia Vale-Costa (282115)

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  9. 1989
    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA targeting ATG9A (siATG9A) for 48 h.

    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA targeting ATG9A (siATG9A) for 48 h. av Sílvia Vale-Costa (282115)

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  10. 1990
    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA non-targeting (siNT) for 48 h.

    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA non-targeting (siNT) for 48 h. av Sílvia Vale-Costa (282115)

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  11. 1991
    Cells (GFP-Rab11a WT<sup>low</sup>) were mock-infected with PR8 virus for 12 h at an MOI of 3.

    Cells (GFP-Rab11a WT<sup>low</sup>) were mock-infected with PR8 virus for 12 h at an MOI of 3. av Sílvia Vale-Costa (282115)

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  12. 1992
    ATG9A distribution is altered during IAV infection.

    ATG9A distribution is altered during IAV infection. av Sílvia Vale-Costa (282115)

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  13. 1993
    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA non-targeting (siNT) for 48 h.

    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA non-targeting (siNT) for 48 h. av Sílvia Vale-Costa (282115)

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  14. 1994
    Functionally active Rab11a is critical for montage of viral inclusions and for viral production.

    Functionally active Rab11a is critical for montage of viral inclusions and for viral production. av Sílvia Vale-Costa (282115)

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  15. 1995
    Cells (A549) were infected with PR8 virus for 12 h, at an MOI of 3.

    Cells (A549) were infected with PR8 virus for 12 h, at an MOI of 3. av Sílvia Vale-Costa (282115)

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  16. 1996
    ATG9A depletion elongates viral inclusions.

    ATG9A depletion elongates viral inclusions. av Sílvia Vale-Costa (282115)

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  17. 1997
    Effect of depleting autophagy factors on mock-infected cells.

    Effect of depleting autophagy factors on mock-infected cells. av Sílvia Vale-Costa (282115)

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  18. 1998
    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA targeting ATG9A (siATG9A) for 48 h.

    Cells (GFP-Rab11a WT<sup>low</sup>, magenta) were treated with siRNA targeting ATG9A (siATG9A) for 48 h. av Sílvia Vale-Costa (282115)

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  19. 1999
    IAV infection impairs Rab11a-regulated recycling and sustains the dynamics of viral inclusions.

    IAV infection impairs Rab11a-regulated recycling and sustains the dynamics of viral inclusions. av Sílvia Vale-Costa (282115)

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  20. 2000
    Cells (GFP-Rab11a DN<sup>low</sup>) were infected with PR8 virus for 12 h, at an MOI of 3.

    Cells (GFP-Rab11a DN<sup>low</sup>) were infected with PR8 virus for 12 h, at an MOI of 3. av Sílvia Vale-Costa (282115)

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