Topology-Driven Mechanical Behavior of Liquid Crystal Elastomers via a Dynamic Mechanical Analyzer حسب Yan-Ting Lin (9345296)

"…Stress relaxation experiments revealed that the synthesized liquid crystal elastomers (LCEs) exhibited significant stress relaxation behavior at 80 °C. Furthermore, when the LCEs were heated to 80 °C without applied stress, their molecular alignment decreased. …"

Clinical and Demographic Characteristics of the Study Population. حسب Káryta Suely Macêdo Martins (22404155)

Baseline and post-treatment results of categorical electrocardiographic variables in each study group. حسب Káryta Suely Macêdo Martins (22404155)

Effects of FSAE in rabbits (n حسب Houkang Ding (20968091)

Baseline and post-treatment results of quantitative electrocardiographic variables in each study group. حسب Káryta Suely Macêdo Martins (22404155)

CONSORT diagram illustrating patient flow during the study. حسب Káryta Suely Macêdo Martins (22404155)

Baseline and Post-Treatment Results of Echocardiographic Variables in Each Experimental Group. حسب Káryta Suely Macêdo Martins (22404155)

Baseline and Post-Treatment Serum Cytokine Levels in Each Investigational Group. حسب Káryta Suely Macêdo Martins (22404155)

OPLS-DA score plot and volcano plot in the control and diarrhea groups. حسب Houkang Ding (20968091)

Baseline and Post-Treatment Serum Levels of NT-proBNP in Each Investigational Group. حسب Káryta Suely Macêdo Martins (22404155)

Variables obtained from the SF-36 quality of life questionnaire in each experimental group. حسب Káryta Suely Macêdo Martins (22404155)

Line graph showing the values of myocardial perfusion defects in the study groups. p = ANOVA Mixed Models. حسب Káryta Suely Macêdo Martins (22404155)

A. Western blot showing accumulation of FoxD3 in CR+DBZ-treated crypt extracts from the colons of Dclk1^ΔIEC mice. حسب Kafayat Yusuf (22104155)

Hazard ratios for Sex across age based on unadjusted (A) and adjusted (B) Cox model. Note: shaded areas representing the 95% confidence intervals; green bars indicating the distrib... حسب Hui Ouyang (245739)

Structural clashes in TP53 mutants: (A) G266E mutation causes steric clashes with PHE109 and LEU257, destabilizing the structure. حسب Ashraf Attia Mahmoud (21262201)

FGF18 injections increase chondroprogenitors and extracellular matrix and reduce chondrocyte hypertrophy after TMJ experimental degeneration. حسب Flavia Gomes Velasque Gama (21175112)

Data Sheet 1_Knockdown of annexin A2 enhances the host cell apoptosis induced by Eimeria tenella.zip حسب Jixia Wang (1685902)

"…Following ANXA2 knockdown, the cell apoptosis rate, caspase-3 activity, and Bax expression levels were significantly increased (P < 0.01), whereas the infection rate and Bcl-2 expression levels were significantly decreased (P < 0.01) compared to the group infected with E. tenella alone. …"

FGF18 injections increase cartilage thickness, chondrocyte proliferation and induce healing after TMJ experimental degeneration. حسب Flavia Gomes Velasque Gama (21175112)

Data Sheet 6_Mechanistic study of the hsa_circ_0074158 binding EIF4A3 impairing sepsis-induced endothelial barrier.zip حسب Haiyan Liao (6100043)

"…RNA pull-down, RNA immunoprecipitation (RIP), and actinomycin D experiments were used to show that circ_0074158 impacts endothelial barrier function in sepsis by reducing the stability of the host gene CTNNA1 (mRNA) after binding to EIF4A3.</p>Results<p>In both LPS-treated human umbilical vein endothelial cells (HUVECs) and cecal ligation and puncture (CLP) murine models, the overexpression of hsa_circ_0074158 (the mouse homolog of hsa_circ_0074158 is named circ_Ctnna1) significantly decreased CTNNA1 mRNA stability and increased endothelial hyperpermeability, while its knockdown restored barrier integrity. …"

Data Sheet 1_Mechanistic study of the hsa_circ_0074158 binding EIF4A3 impairing sepsis-induced endothelial barrier.docx حسب Haiyan Liao (6100043)

"…RNA pull-down, RNA immunoprecipitation (RIP), and actinomycin D experiments were used to show that circ_0074158 impacts endothelial barrier function in sepsis by reducing the stability of the host gene CTNNA1 (mRNA) after binding to EIF4A3.</p>Results<p>In both LPS-treated human umbilical vein endothelial cells (HUVECs) and cecal ligation and puncture (CLP) murine models, the overexpression of hsa_circ_0074158 (the mouse homolog of hsa_circ_0074158 is named circ_Ctnna1) significantly decreased CTNNA1 mRNA stability and increased endothelial hyperpermeability, while its knockdown restored barrier integrity. …"