Effect of parasitism and PDV injection on triglyceride concentration and lipid metabolism-related gene expression in FAW larvae. by Xian Li (201841)

Effect of parasitism and PDV injection on lipid droplet size in the fat body of FAW larvae. by Xian Li (201841)

<i>rfbV</i> gene triplication in S. Paratyphi A increases VL O-ag production and reduces C3b deposition. by Fermin E. Guerra (6084545)

“…Statistical analysis was performed by one sample t test with a hypothetical value of 1.0, with P values in red indicating statistical significance as P < 0.05. Column bars represent the mean with error bars showing standard deviation. …”

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. by Erienne G. Norton (9612079)

Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. by Erienne G. Norton (9612079)

“…See also <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3003074#pbio.3003074.s017" target="_blank">S9 Table</a>. Data are shown as mean ± s.e.m. (<b>D</b>). Wilcoxon rank-sum test (<b>A</b>, <b>B</b>; <b>J</b>, right), Benjamini–Hochbert test (<b>C</b>), two-tailed Student <i>t</i> test (<b>D</b>, <b>I</b>), Fisher’s exact test (<b>F</b>–<b>H</b>), or bionomial test (<b>J</b>, left); NS, not significant. …”

ASFV-ΔI7L induces higher immune responses than does ASFV-WT in pigs. by Meilin Li (1832905)

Differential expression profiling in the ASFV-ΔI7L-infected primary porcine alveolar macrophages (PAMs) by RNA sequencing analysis. by Meilin Li (1832905)

Effect of <i>CrBV3-31</i> interference on lipid metabolism in FAW larvae. by Xian Li (201841)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. by Erienne G. Norton (9612079)

pI7L suppresses the IFN-γ-triggered JAK-STAT signaling pathway. by Meilin Li (1832905)

The <i>I7L</i> gene is involved in ASFV pathogenicity in pigs. by Meilin Li (1832905)

Effect of <i>CrBV3-31</i> interference on the performance of <i>Cotesia ruficrus</i> offspring. by Xian Li (201841)

The biological characteristics of the I7L protein (pI7L). by Meilin Li (1832905)

Summary of related studies. by Most. Fatematuz Zohora (19429152)

“…To address these challenges, we propose the Dynamic Banker’s Deadlock Avoidance Algorithm (DBDAA). …”

Time complexity of different safety algorithms. by Most. Fatematuz Zohora (19429152)

“…To address these challenges, we propose the Dynamic Banker’s Deadlock Avoidance Algorithm (DBDAA). …”

Process execution halted due to deadlock. by Most. Fatematuz Zohora (19429152)

“…To address these challenges, we propose the Dynamic Banker’s Deadlock Avoidance Algorithm (DBDAA). …”

Novel Small-Molecule miR-124 Inducer Acts as “a Physiological Brake” of Inflammation in Ulcerative Colitis by Targeting the PIK3R2/PI3K/Akt Axis by Tiantian Wang (1688881)

“…This study identified FHND5032, a novel small-molecule miR-124 inducer, as a potent therapeutic candidate for UC. We found that FHND5032 significantly upregulated miR-124 expression in macrophages, surpassing the clinical-stage comparator ABX464 in vitro and in vivo. …”

Discovery of an Orally Active PDE1 Inhibitor for Disease-Modifying Treatment of Postmenopausal Osteoporosis via Dual Anabolic-Antiresorptive Mechanisms by Farman M. Abbasi (22318766)

“…Postmenopausal osteoporosis (PMO) is characterized by an imbalance in bone remodeling with increased osteoclast and decreased osteoblast activity, leading to bone loss and higher fracture risk. …”

Discovery of an Orally Active PDE1 Inhibitor for Disease-Modifying Treatment of Postmenopausal Osteoporosis via Dual Anabolic-Antiresorptive Mechanisms by Farman M. Abbasi (22318766)

“…Postmenopausal osteoporosis (PMO) is characterized by an imbalance in bone remodeling with increased osteoclast and decreased osteoblast activity, leading to bone loss and higher fracture risk. …”

Discovery of an Orally Active PDE1 Inhibitor for Disease-Modifying Treatment of Postmenopausal Osteoporosis via Dual Anabolic-Antiresorptive Mechanisms by Farman M. Abbasi (22318766)

“…Postmenopausal osteoporosis (PMO) is characterized by an imbalance in bone remodeling with increased osteoclast and decreased osteoblast activity, leading to bone loss and higher fracture risk. …”