Box plots to show various parameters of the rectal waves ED, HFD and SDG larvae compared with the SD controls, at 7 dpf. by Jessica C. M. Hui (20370522)

Box plots to show various parameters of GI motility determined from AB larvae at 7 dpf that were fed a standard diet (SD). by Jessica C. M. Hui (20370522)

Role of MIB2 and CAMK2 in FAT1-depndent regulation of YAP/TAZ protein levels and YAP/TAZ nuclear localization. by Rui Li (4631)

Regulation of YAP/TAZ through MIB2 in HNSCCs. by Rui Li (4631)

Graphs showing the qPCR data from SD, ED, HFD and SDG larvae at 7 dpf (n = 6 for each). by Jessica C. M. Hui (20370522)

Quantification of fasting glucose, random glucose, and insulin levels. by Jessica C. M. Hui (20370522)

E3 ligase TRIM56 is up-regulated in response to CVB3 infection. by Yao Wang (102387)

Measurements of body mass index (BMI) and adipose deposits. by Jessica C. M. Hui (20370522)

Loss of FAT1 expression results in increased YAP/TAZ protein levels. by Rui Li (4631)

Mas Receptor Phosphorylation and its-mediated Effects in Renal Cortex of WKY Rats and SHRs. by Lin Chen (54305)

S1 File - by Kimesh Loganathan Naidoo (19697486)

“…Of the 45 015 admissions analysed, 1237(2·75%) demised with significant decreases in admissions during all the lockdown levels, with the most significant mean monthly decrease of 450(95%, CI = 657·3, -244·3) p<0·001 in level 5 (the most severe) lockdown. …”

Role of YAP/TAZ in FAT1-regulated HNSCC growth. by Rui Li (4631)

Box plot of queens and males in the young colony (left) and old colony (right) A-B compares the queens and males distribution in the young and old colonies, respectively. by Moïse Pierre Exélis (21578515)

The phosphorylation of Mas receptor and its induced-effects in <i>hGRK4</i> <i>γ</i> WT and 142V transgenic mice. by Lin Chen (54305)

FAT1 and MIB2 control tumor growth <i>in vivo.</i> by Rui Li (4631)

TRIM56 promotes the ubiquitination and proteasomal degradation of CVB3 3D. by Yao Wang (102387)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. by Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. by Erienne G. Norton (9612079)

Atg14 does not contribute to terminal eTreg generation during perinatal life. by Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. by Erienne G. Norton (9612079)

“…Relative (normalized to average control in each experiment) fold-change comparisons between genotypes in each cell type are shown in bold (<b>A</b>, <b>B</b>). Data are shown as mean ±  s.e.m. (<b>A</b>, <b>B</b>). Two-tailed Student <i>t</i> test (<b>A</b>, <b>B</b>, <b>I</b>), Benjamini–Hochberg test (<b>C</b>), Wilcoxon rank-sum test (<b>G</b>), Fisher’s exact tes<i>t</i> (<b>H</b>), or binomial test (<b>J</b>); NS, not significant. …”