Overexpression of TNFAIP9 alleviated the renal fibrosis caused by 5/6 Nx in C57 mice. by Ying Chen (9697)

Overexpression of TNFAIP9 remitted the renal inflammation caused by 5/6 Nx in C57 mice. by Ying Chen (9697)

Overexpression of TNFAIP9 suppressed inflammation in mouse peritoneal macrophages caused by 5/6 Nx in C57 mice. by Ying Chen (9697)

Overexpression of TNFAIP9 inhibited the TGF-β-induced fibrosis in HK-2 cells. by Ying Chen (9697)

TNFAIP9 expression was increased in mice with chronic kidney disease (CKD) induced by 5/6 nephrectomy (Nx). by Ying Chen (9697)

Overexpression of TNFAIP9 counteracted the renal damage caused by 5/6 Nx in C57 mice. by Ying Chen (9697)

Threading Behavior and Dynamics of Ring-Linear Polymer Blends under Poiseuille Flow by Deyin Wang (6028850)

“…However, as rings are semiflexible (<i>K</i>_br > 0), the threading probability significantly decreases and then significantly increases as the flow field strength increases. …”

S1_raw_images. by Lijun Mao (561880)

“…However, the involvement of miR-122 in regulation of TGF-β signaling and its implication in TGF-β-related HCC metastasis remains obscure. In this study, we demonstrated that miR-122 significantly enhanced the activities of the TGF-β pathway reporter, the levels of phosphorylation of Smad2 and Smad3, and the expression of mesenchymal markers (N-cadherin and vimentin) in HCC cells. …”

Raw data for graphs. by Lijun Mao (561880)

“…However, the involvement of miR-122 in regulation of TGF-β signaling and its implication in TGF-β-related HCC metastasis remains obscure. In this study, we demonstrated that miR-122 significantly enhanced the activities of the TGF-β pathway reporter, the levels of phosphorylation of Smad2 and Smad3, and the expression of mesenchymal markers (N-cadherin and vimentin) in HCC cells. …”

The predicted AREs in the 3’UTR of ITGAV mRNA. by Lijun Mao (561880)

“…However, the involvement of miR-122 in regulation of TGF-β signaling and its implication in TGF-β-related HCC metastasis remains obscure. In this study, we demonstrated that miR-122 significantly enhanced the activities of the TGF-β pathway reporter, the levels of phosphorylation of Smad2 and Smad3, and the expression of mesenchymal markers (N-cadherin and vimentin) in HCC cells. …”

Overexpression of TNFAIP9 inhibited the TNF-α-induced inflammation in HK-2 cells. by Ying Chen (9697)

S1 Data - by Bowen Deng (3123237)

“…</p><p>Results</p><p>Among 1,523 eligible patients, 227 (14.9%) died within 30 days. We identified 4 longitudinal trajectories of blood glucose, including class 1 (a low initial average blood glucose level with steady trend within 24 hours), class 2 (a high initial average blood glucose with gently decreased trend), class 3 (the highest initial average blood glucose with rapidly decreased trend) and class 4 (a high initial average blood glucose level with the trend that increased at first and then decreased). …”

Characteristics of AMI patients with DM. by Bowen Deng (3123237)

“…</p><p>Results</p><p>Among 1,523 eligible patients, 227 (14.9%) died within 30 days. We identified 4 longitudinal trajectories of blood glucose, including class 1 (a low initial average blood glucose level with steady trend within 24 hours), class 2 (a high initial average blood glucose with gently decreased trend), class 3 (the highest initial average blood glucose with rapidly decreased trend) and class 4 (a high initial average blood glucose level with the trend that increased at first and then decreased). …”

Molecular dynamics simulation results for Apo-TP53 (red), wild-typeTP53. by Ashraf Attia Mahmoud (21262201)

PP2B dephosphorylates pS295 <i>in vitro</i>. by Takahiko Chimura (729098)

Influx of extracellular calcium activates PP2B. by Takahiko Chimura (729098)

PP2B, but not PP1/PP2A, is involved in the NMDA-induced dephosphorylation of pS295. by Takahiko Chimura (729098)

Factors associated with the <i>SS</i> Positives. by Anuradha Rajamanickam (5278013)

“…<div><p><i>Strongyloides stercoralis (Ss)</i> is a parasitic infection affecting 50–100 million people globally, with significant immune and metabolic consequences, particularly in immunocompromised individuals. …”

PP1/PP2A does not dephosphorylate pS295 <i>in vitro</i>. by Takahiko Chimura (729098)

“…The data are represented as the mean ± standard deviation (n = 3 from three independent experiments). …”

The PP1/PP2A inhibitor calyculin A increases phosphorylated Ser295 of PSD-95 concomitantly with the activation of JNK1. by Takahiko Chimura (729098)