The injectable contraceptives depot medroxyprogesterone acetate and norethisterone enanthate substantially and differentially decrease testosterone and sex hormone binding globulin... by Chanel Avenant (409756)

“…<p dir="ltr">HIV acquisition risk with norethisterone (NET) enanthate (NET-EN) is reportedly less than for depo-medroxyprogesterone acetate intramuscular (DMPA-IM). We investigated the effects of these progestin-only injectable contraceptives on serum testosterone and sex hormone binding globulin (SHBG) levels, since these may play a role in sexual behavior and HIV acquisition. …”

S1 File - by Changxu Wang (17826805)

Subjects:

The flow chart of the study. by Changxu Wang (17826805)

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Comparison of postoperative NLR, PLR, and SII between the two groups. by Changxu Wang (17826805)

Subjects:

Initial preparation state diagram of the specimen experiment. by Jia Zhu (135506)

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Overexpression of MALS-1 reduces mitochondria in the PLM axon and causes axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

“…<p>(A–C) Overexpressed MALS-1::Scarlet protein colocalizes with mitochondria. …”

Loss of MALS-1 function suppresses the mitochondrial and axon degeneration phenotypes that are caused by loss of RBM-26 function. by Tamjid A. Chowdhury (14149846)

“…For panel B, error bars represent the standard error of the mean (<i>n</i> = 25), and statistical significance was analyzed by one-way ANOVA with a Tukey post hoc test (*<i>p</i> < 0.05 and **<i>p</i> < 0.01). …”

Graphs showing the temporal effects of temperature at the four different positions during a drilling sequence with guide drilling in two steps and final widening drilling (MIPS) an... by Marsel Ganeyev (21453399)

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RBM-26 binds to the <i>mals-1</i> mRNA and negatively regulates its expression. by Tamjid A. Chowdhury (14149846)

RBM-26 is expressed in the PLM and functions within neurons to protect against axon degeneration and prevent axon overlap defects. by Tamjid A. Chowdhury (14149846)

Mutations in <i>rbm-26</i> cause axon degeneration and axon overlap defects. by Tamjid A. Chowdhury (14149846)

Expression of RBM-26 is disrupted by P80L and L13V missense mutations. by Tamjid A. Chowdhury (14149846)

All data points from Fig 2. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 5. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 8. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 3. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 1. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 4. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 9. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 7. by Sara Hijazi (21656615)

“…Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”