Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. by Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. by Erienne G. Norton (9612079)

“…(<b>I</b>) Quantification of Bim/Mcl1 (left) and Bim/Bcl2 (right) ratios (based on gMFIs) from TCRβ⁺CD4⁺Foxp3⁺CD44^lowCD62L^high cTregs or TCRβ⁺CD4⁺Foxp3⁺CD44^highCD62L^low eTregs derived from the spleen of control (<i>n</i> = 19) or <i>Foxp3</i>^Cre<i>Pik3c3</i>^fl/fl (<i>n</i> = 17) mixed BM chimera mice. gMFI, geometric mean fluorescence intensity. Data are shown as mean ± s.e.m. …”

Atg14 does not contribute to terminal eTreg generation during perinatal life. by Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. by Erienne G. Norton (9612079)

“…Relative (normalized to average control in each experiment) fold-change comparisons between genotypes in each cell type are shown in bold (<b>A</b>, <b>B</b>). Data are shown as mean ±  s.e.m. (<b>A</b>, <b>B</b>). Two-tailed Student <i>t</i> test (<b>A</b>, <b>B</b>, <b>I</b>), Benjamini–Hochberg test (<b>C</b>), Wilcoxon rank-sum test (<b>G</b>), Fisher’s exact tes<i>t</i> (<b>H</b>), or binomial test (<b>J</b>); NS, not significant. …”

Atg14 plays a cell-intrinsic role in orchestrating eTreg survival but not terminal differentiation. by Erienne G. Norton (9612079)

“…Jun/Fos transcription factors with predicted decreased activity in Atg14-deficient versus control eTregs are labeled. …”

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. by Erienne G. Norton (9612079)

Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. by Erienne G. Norton (9612079)

“…See also <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3003074#pbio.3003074.s017" target="_blank">S9 Table</a>. Data are shown as mean ± s.e.m. (<b>D</b>). Wilcoxon rank-sum test (<b>A</b>, <b>B</b>; <b>J</b>, right), Benjamini–Hochbert test (<b>C</b>), two-tailed Student <i>t</i> test (<b>D</b>, <b>I</b>), Fisher’s exact test (<b>F</b>–<b>H</b>), or bionomial test (<b>J</b>, left); NS, not significant. …”

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. by Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. by Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. by Erienne G. Norton (9612079)

Variation of shear stress during the pullout of a single fiber with respect to the shear strain [41]. by Ping Gao (172038)

Expression of 20 CrBV genes in different tissues of FAW following parasitism by <i>Cotesia ruficrus.</i> by Xian Li (201841)

Expression of 20 CrBV genes in FAW at different time points following parasitism by <i>Cotesia ruficrus.</i> by Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV3–31</i> interference. by Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV13–7</i> interference. by Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV11–6</i> interference. by Xian Li (201841)

GO and KEGG analysis of DEPs. by Huiyang Jia (14444661)

Effect of parasitism and PDV injection on triglyceride concentration and lipid metabolism-related gene expression in FAW larvae. by Xian Li (201841)

Effect of parasitism and PDV injection on lipid droplet size in the fat body of FAW larvae. by Xian Li (201841)

Effect of O-ag modifying glycosyltransferases on complement activation and <i>Salmonella</i> interactions with human serum. by Fermin E. Guerra (6084545)

“…Statistical analysis was performed by one-way ANOVA on 3–4 biological replicates, with P values in red indicating statistical significance with P < 0.05. Column bars represent the means, with error bars displaying standard deviation. …”