Vps34 orchestrates Treg function in vivo. حسب Erienne G. Norton (9612079)

<i>rfbV</i> gene triplication in S. Paratyphi A increases VL O-ag production and reduces C3b deposition. حسب Fermin E. Guerra (6084545)

"…Statistical analysis was performed by one sample t test with a hypothetical value of 1.0, with P values in red indicating statistical significance as P < 0.05. Column bars represent the mean with error bars showing standard deviation. …"

Estimating the transportation cost matrix from microstate joint probability of consecutive timesteps at rest. حسب Giacomo Barzon (19832364)

Targeting of Vps34 reduces B16F10 tumor growth and working model for Vps34-dependent regulation of eTreg transitional heterogeneity and maintenance. حسب Erienne G. Norton (9612079)

Loss of Vps34 reduces Treg suppressive function and is associated with the development of type-I-dominant inflammatory disease. حسب Erienne G. Norton (9612079)

Vps34 supports Treg maintenance in lymphoid and non-lymphoid tissues. حسب Erienne G. Norton (9612079)

"…(<b>I</b>) Quantification of Bim/Mcl1 (left) and Bim/Bcl2 (right) ratios (based on gMFIs) from TCRβ⁺CD4⁺Foxp3⁺CD44^lowCD62L^high cTregs or TCRβ⁺CD4⁺Foxp3⁺CD44^highCD62L^low eTregs derived from the spleen of control (<i>n</i> = 19) or <i>Foxp3</i>^Cre<i>Pik3c3</i>^fl/fl (<i>n</i> = 17) mixed BM chimera mice. gMFI, geometric mean fluorescence intensity. Data are shown as mean ± s.e.m. …"

Minimal data set. حسب Daan P. van den Brink (9900073)

"…This effect might be attributed to decreased resuscitation volumes rather than the type of fluid.…"

Atg14 does not contribute to terminal eTreg generation during perinatal life. حسب Erienne G. Norton (9612079)

Chromatin accessibility analysis of perinatal Vps34-deficient Tregs. حسب Erienne G. Norton (9612079)

"…Relative (normalized to average control in each experiment) fold-change comparisons between genotypes in each cell type are shown in bold (<b>A</b>, <b>B</b>). Data are shown as mean ±  s.e.m. (<b>A</b>, <b>B</b>). Two-tailed Student <i>t</i> test (<b>A</b>, <b>B</b>, <b>I</b>), Benjamini–Hochberg test (<b>C</b>), Wilcoxon rank-sum test (<b>G</b>), Fisher’s exact tes<i>t</i> (<b>H</b>), or binomial test (<b>J</b>); NS, not significant. …"

Atg14 plays a cell-intrinsic role in orchestrating eTreg survival but not terminal differentiation. حسب Erienne G. Norton (9612079)

"…Jun/Fos transcription factors with predicted decreased activity in Atg14-deficient versus control eTregs are labeled. …"

Analysis of Tregs from perinatal mice and mixed BM chimeras for transitional and terminal eTreg states. حسب Erienne G. Norton (9612079)

Vps34 orchestrates transcriptional and epigenetic remodeling events corresponding to terminal eTreg differentiation during perinatal life. حسب Erienne G. Norton (9612079)

"…See also <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3003074#pbio.3003074.s017" target="_blank">S9 Table</a>. Data are shown as mean ± s.e.m. (<b>D</b>). Wilcoxon rank-sum test (<b>A</b>, <b>B</b>; <b>J</b>, right), Benjamini–Hochbert test (<b>C</b>), two-tailed Student <i>t</i> test (<b>D</b>, <b>I</b>), Fisher’s exact test (<b>F</b>–<b>H</b>), or bionomial test (<b>J</b>, left); NS, not significant. …"

Uvrag in Tregs is dispensable for establishment of immune tolerance and eTreg accumulation. حسب Erienne G. Norton (9612079)

Targeting of Vps34 or Atg14, but not Uvrag, in Tregs reduces tumor growth in mouse models. حسب Erienne G. Norton (9612079)

Atg14 deficiency in Tregs induces a type-I-dominant lymphoproliferative disease and cell-intrinsic reductions in eTregs. حسب Erienne G. Norton (9612079)

Variation of shear stress during the pullout of a single fiber with respect to the shear strain [41]. حسب Ping Gao (172038)

Expression of 20 CrBV genes in different tissues of FAW following parasitism by <i>Cotesia ruficrus.</i> حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW at different time points following parasitism by <i>Cotesia ruficrus.</i> حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV3–31</i> interference. حسب Xian Li (201841)

Expression of 20 CrBV genes in FAW following <i>CrBV13–7</i> interference. حسب Xian Li (201841)