Intraperitoneal injection protocol for inhibitors (A), inhibitors DAS and Y27632 reduced intestinal permeability (B) and intestinal adult worm burden (C) of infected mice. by Wen Wen Zheng (17721404)

Fig 6 - by Teagan McMahon (20108036)

Proliferation and differentiation of myoblasts derived from satellite cells in WT and NMIIC KO mice. by Hiroki Hamaguchi (22783780)

Phenotypic characterization of NMIIC knockout (KO) mice. by Hiroki Hamaguchi (22783780)

PRV UL4 augments ASC oligomerization and speck formation. by Xiaohua Zhang (350570)

Effects of allopurinol on chronic PG-LPS-induced oxidative stress in cardiac muscle. by Akinaka Morii (12709672)

Effects of allopurinol on cardiac myocyte apoptosis induced by chronic PG-LPS infusion. by Akinaka Morii (12709672)

Effects of allopurinol on PG-LPS-induced fibrosis in cardiac muscle. by Akinaka Morii (12709672)

Differences in mean scores of upper extremity performance in all five accelerometer-derived variables across four time points- pre-HABIT, post-HABIT, 3-month, and 6-month post-HABI... by Shailesh S. Gardas (20490020)

“…<p>Values reported are means ± SEM as determined by distribution of data during each assessment time point. …”

Characteristics of the participants. by Kensaku Sasayama (741571)

“…Overall physical activity levels decreased at follow-up compared to baseline. However, the contribution of commuting school activities to overall physical activity significantly increased at follow-up compared to baseline, especially in step counts and MVPA. …”

Fig 2 - by Nathalie Lavenne-Collot (11262998)

Supplementary file 1_Bone marrow adipose tissue expansion and bone loss in experimental chronic kidney disease is independent of altered bone marrow stromal cell lineage determinat... by Worachet Promruk (22286701)

“…Introduction<p>Chronic kidney disease-mineral bone disorder is the irreversible loss of kidney function leading to altered mineral homeostasis and bone loss, commonly referred to as renal osteodystrophy. …”

All-Atom Simulations Reveal the Effect of Membrane Composition on the Signaling of the NKG2A/CD94/HLA‑E Immune Receptor Complex by Martin Ljubič (15680161)

“…Furthermore, the DPPC model with a membrane possessing a high transition temperature in a gel-like state became curved, affecting the exposure of one ITIM region. The decreased membrane thickness in the DPLC model caused a significant transmembrane domain tilt, altering the linker protrusion angle and potentially disrupting the hydrogen bonding network in the extracellular domain. …”

All data points from Fig 2. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 5. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 8. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 3. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 1. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 4. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”

All data points from Fig 9. by Sara Hijazi (21656615)

“…Using whole-cell patch-clamp recordings to examine intrinsic properties of PV interneurons in the PFC, we found that juvenile demyelination in mice induced robust alterations of PV interneuron firing patterns. Specifically, we observed that demyelination caused an impairment in the ability of PV interneurons to sustain high-frequency firing associated with a substantial decrease in Kv3-specific currents. …”