Familial molar tissues due to mutations in the inflammatory gene, NALP7, have normal postzygotic DNA methylation

An imprinting disorder has been believed to underlie the etiology of familial biparental hydatidiform moles (HMs) based on the abnormal methylation or expression of imprinted genes in molar tissues. However, the extent of the epigenetic defect in these tissues and the developmental stage at which th...

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Bibliographic Details
Main Author: El Maarri, Osman (author)
Other Authors: Djuric, Ugljesa (author), Lamb, Barbara (author), Kuick, Rork (author), Seoud, Muheiddine (author), Coullin, Philippe (author), Oldenburg, Johannes (author), Hanash, Samir (author), Slim, Rima (author)
Format: article
Published: 2006
Online Access:http://hdl.handle.net/10725/6176
http://dx.doi.org/10.1007/s00439-006-0192-0
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://link.springer.com/article/10.1007/s00439-006-0192-3
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