Familial molar tissues due to mutations in the inflammatory gene, NALP7, have normal postzygotic DNA methylation

An imprinting disorder has been believed to underlie the etiology of familial biparental hydatidiform moles (HMs) based on the abnormal methylation or expression of imprinted genes in molar tissues. However, the extent of the epigenetic defect in these tissues and the developmental stage at which th...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: El Maarri, Osman (author)
مؤلفون آخرون: Djuric, Ugljesa (author), Lamb, Barbara (author), Kuick, Rork (author), Seoud, Muheiddine (author), Coullin, Philippe (author), Oldenburg, Johannes (author), Hanash, Samir (author), Slim, Rima (author)
التنسيق: article
منشور في: 2006
الوصول للمادة أونلاين:http://hdl.handle.net/10725/6176
http://dx.doi.org/10.1007/s00439-006-0192-0
http://libraries.lau.edu.lb/research/laur/terms-of-use/articles.php
https://link.springer.com/article/10.1007/s00439-006-0192-3
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