DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)

DJ-1 is a 20-KDa protein that was first identified as an oncogene product responsible for a subset of familial Parkinson’s disease; hence its name PARK 7. Previous studies showed that DJ-1 negatively regulated the tumor suppressor gene PTEN expression; thus promoting the phosphorylation of the PI3K/...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Yassine, Rawan M. (author)
التنسيق: masterThesis
منشور في: 2016
الموضوعات:
الوصول للمادة أونلاين:http://hdl.handle.net/10725/3258
https://doi.org/10.26756/th.2014.62
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_version_ 1864513460923006976
author Yassine, Rawan M.
author_facet Yassine, Rawan M.
author_role author
dc.creator.none.fl_str_mv Yassine, Rawan M.
dc.date.none.fl_str_mv 2016-03-03T13:17:44Z
2016-03-03T13:17:44Z
2016-03-03
5/16/2014
dc.identifier.none.fl_str_mv http://hdl.handle.net/10725/3258
https://doi.org/10.26756/th.2014.62
dc.language.none.fl_str_mv en
dc.publisher.none.fl_str_mv Lebanese American University
dc.rights.*.fl_str_mv info:eu-repo/semantics/openAccess
dc.subject.none.fl_str_mv Rho GTPases
Cells -- Motility
Antioncogenes
Lebanese American University -- Dissertations
Dissertations, Academic
dc.title.none.fl_str_mv DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
dc.type.none.fl_str_mv Thesis
info:eu-repo/semantics/publishedVersion
info:eu-repo/semantics/masterThesis
description DJ-1 is a 20-KDa protein that was first identified as an oncogene product responsible for a subset of familial Parkinson’s disease; hence its name PARK 7. Previous studies showed that DJ-1 negatively regulated the tumor suppressor gene PTEN expression; thus promoting the phosphorylation of the PI3K/Akt signaling pathway, and activating cell proliferation and transformation. Therefore, DJ-1 can be used as an indication of cancer metastasis and can be a potential therapeutic target. However, the localization, detailed mechanism and the role of DJ-1 in cellular motility are still not fully understood. Therefore, our study showed that DJ-1, at normal cellular condition, is present in the nucleus, mitochondria, golgi apparatus and plasma membrane of astrocytoma (SF268) cells. Upon its stimulation with EGF, DJ-1 localize outside the nucleus, leading to an increase in cellular motility in lung, brain, and breast cancer cells by activating the PI3K pathway. We also showed that this activation is carried out by inhibiting PTEN and thus regulating the RhoGTPases mainly Rac which is known to be involved in cellular motility.
eu_rights_str_mv openAccess
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network_acronym_str LAURepo
network_name_str Lebanese American University repository
oai_identifier_str oai:laur.lau.edu.lb:10725/3258
publishDate 2016
publisher.none.fl_str_mv Lebanese American University
repository.mail.fl_str_mv
repository.name.fl_str_mv
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spelling DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)Yassine, Rawan M.Rho GTPasesCells -- MotilityAntioncogenesLebanese American University -- DissertationsDissertations, AcademicDJ-1 is a 20-KDa protein that was first identified as an oncogene product responsible for a subset of familial Parkinson’s disease; hence its name PARK 7. Previous studies showed that DJ-1 negatively regulated the tumor suppressor gene PTEN expression; thus promoting the phosphorylation of the PI3K/Akt signaling pathway, and activating cell proliferation and transformation. Therefore, DJ-1 can be used as an indication of cancer metastasis and can be a potential therapeutic target. However, the localization, detailed mechanism and the role of DJ-1 in cellular motility are still not fully understood. Therefore, our study showed that DJ-1, at normal cellular condition, is present in the nucleus, mitochondria, golgi apparatus and plasma membrane of astrocytoma (SF268) cells. Upon its stimulation with EGF, DJ-1 localize outside the nucleus, leading to an increase in cellular motility in lung, brain, and breast cancer cells by activating the PI3K pathway. We also showed that this activation is carried out by inhibiting PTEN and thus regulating the RhoGTPases mainly Rac which is known to be involved in cellular motility.N/A1 hard copy: xi, 55 leaves; ill.; 30 cm. available at RNL.Includes bibliographical references (leaves 43-55).Lebanese American University2016-03-03T13:17:44Z2016-03-03T13:17:44Z5/16/20142016-03-03Thesisinfo:eu-repo/semantics/publishedVersioninfo:eu-repo/semantics/masterThesishttp://hdl.handle.net/10725/3258https://doi.org/10.26756/th.2014.62eninfo:eu-repo/semantics/openAccessoai:laur.lau.edu.lb:10725/32582023-11-08T09:47:18Z
spellingShingle DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
Yassine, Rawan M.
Rho GTPases
Cells -- Motility
Antioncogenes
Lebanese American University -- Dissertations
Dissertations, Academic
status_str publishedVersion
title DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
title_full DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
title_fullStr DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
title_full_unstemmed DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
title_short DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
title_sort DJ-1, a peten inhibitor, is a positive regulator of cancer cell motility. (c2014)
topic Rho GTPases
Cells -- Motility
Antioncogenes
Lebanese American University -- Dissertations
Dissertations, Academic
url http://hdl.handle.net/10725/3258
https://doi.org/10.26756/th.2014.62