Role of microRNA 690 in Mediating Angiotensin II Effects on Inflammation and Endoplasmic Reticulum Stress

<p dir="ltr">Overactivation of the renin–angiotensin system (RAS) during obesity disrupts adipocyte metabolic homeostasis and induces endoplasmic reticulum (ER) stress and inflammation; however, underlying mechanisms are not well known. We propose that overexpression of angiotensinog...

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محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Kalhara R. Menikdiwela (18566698) (author)
مؤلفون آخرون:	Latha Ramalingam (18300808) (author), Mostafa M. Abbas (17058093) (author), Halima Bensmail (10400) (author), Shane Scoggin (18566701) (author), Nishan S. Kalupahana (18566704) (author), Asha Palat (18566707) (author), Preethi Gunaratne (8410134) (author), Naima Moustaid-Moussa (66960) (author)
منشور في:	2020
الموضوعات:	Biological sciences Biochemistry and cell biology Genetics Biomedical and clinical sciences Metabolic medicine adipocytes renin–angiotensin system (RAS) obesity miR-690 endoplasmic reticulum (ER) stress inflammation
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Role of microRNA 690 in Mediating Angiotensin II Effects on Inflammation and Endoplasmic Reticulum Stress

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