DNAJB3/HSP-40 cochaperone improves insulin signaling and enhances glucose uptake in vitro through JNK repression

<p dir="ltr">Heat shock response (HSR) is an essential host-defense mechanism that is dysregulated in obesity-induced insulin resistance and type 2 diabetes (T2D). Our recent data demonstrated that DNAJB3 was downregulated in obese human subjects and showed negative correlation with...

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التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Mohamed Abu-Farha (25322) (author)
مؤلفون آخرون:	Preethi Cherian (437070) (author), Irina Al-Khairi (437069) (author), Ali Tiss (437068) (author), Abdelkrim Khadir (437074) (author), Sina Kavalakatt (437073) (author), Samia Warsame (437075) (author), Mohammed Dehbi (309033) (author), Kazem Behbehani (412126) (author), Jehad Abubaker (437067) (author)
منشور في:	2015
الموضوعات:	Biomedical and clinical sciences Medical biochemistry and metabolomics Heat Shock Response (HSR) Insulin Resistance Type 2 Diabetes (T2D) DNAJB3 Obesity Inflammatory Markers Gene Expression
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DNAJB3/HSP-40 cochaperone improves insulin signaling and enhances glucose uptake in vitro through JNK repression

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