Raw data of the charts in Fig 5.

<div><p>Epstein-Barr virus (EBV) manipulates the ubiquitin-proteasome system and regulators of Bcl-2 family to enable the persistence of the virus and survival of the host cells through the expression of viral proteins in distinct latency patterns. We postulate that the combination of bo...

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Bibliographic Details
Main Author:	Kam Pui Tam (19746865) (author)
Other Authors:	Jia Xie (247149) (author), Rex Kwok Him Au-Yeung (19746868) (author), Alan K. S. Chiang (8677455) (author)
Published:	2024
Subjects:	Biochemistry Medicine Microbiology Cell Biology Molecular Biology Pharmacology Immunology Cancer Mental Health Hematology Infectious Diseases Virology transplant lymphoproliferative disorder latent membrane protein distinct latency patterns cause synergistic killing b signaling pathway associated hemophagocytic lymphohistiocytosis nf -&# 954 b &# 945 bortezomib potently induced xlink "> epstein latent viral proteins ebv nuclear antigen cell cycle arrest bortezomib prevented lmp 2 causing g1 venetoclax inhibited bcl viral proteins &# 954 inhibited mcl venetoclax targets venetoclax decreased taken together scid mice proteasome system proteasome inhibitor phosphorylated p65 host cells either lmp dependent manner data corroborated cyclin d1 apoptotic initiator 2 upregulated 2 simultaneously 2 inhibitor 2 family 001 ).
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Raw data of the charts in Fig 5.

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