PIM1 overexpression promotes senescence in L929 cells.

<p>(A)PIM1 mRNA expression in L929 cells transfected with the PIM1 overexpression plasmid was detected using RT-qPCR. (B)Expression and quantitative analysis of PIM1 protein in L929 cells transfected with the PIM1 overexpression plasmid were determined by Western blot. (C) Representative image...

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Bibliographic Details
Main Author:	Ya Xiao (691453) (author)
Other Authors:	Mao Chen (92698) (author), Lingyun Li (142918) (author), Liying Chen (4223026) (author), Xiaoyu Tian (1889653) (author), Xiaoyu Huang (24573) (author), Fangyi Zhu (1416895) (author), Bingshu Li (4335934) (author), Li Hong (426614) (author)
Published:	2025
Subjects:	Biochemistry Cell Biology Molecular Biology Physiology Pharmacology Biotechnology Immunology Developmental Biology Marine Biology Cancer Infectious Diseases vitro experiments involved stress urinary incontinence impairing dna repair associated heterochromatin foci vaginal balloon dilation senescence pathogenic pathway related fibroblast senescence mediated cellular senescence induced cellular senescence treated fibroblasts subjected 1 &# 945 pim1 facilitated senescence study identifies pim1 pim1 inhibitor azd pim1 induces hypoxia cellular senescence vaginal wall 1 staining &# 947 senescence markers senescence burden pim1 knockdown pim1 kinase pim1 inhibition study revealed study aims xlink "> x accumulation utilizing hypoxyprobetm two pathways therapeutic potential significantly elevated significant improvements potential target postpartum prevention particular relevance novel approach notable upregulation mouse model ki67 expression investigate hypoxia hypoxia markers functional outcomes findings elucidate expression analysis effectively reversed comprehensive hypoxia bladder function
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PIM1 overexpression promotes senescence in L929 cells.

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