SH2D5 knockdown inhibits lung adenocarcinoma (LUAD) cell proliferation, migration and invasion.

<p>(A, B) The western blot analysis was employed to detect SH2D5 knockdown efficiency of transfection in H1299 cells. (C, D) The CCK8 and colony formation assays were used to reveal the cell proliferation in SH2D5- knockdown H1299 cells. (E F) Wound healing and transwell assays were used to de...

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Bibliographic Details
Main Author:	Licheng Du (16469836) (author)
Other Authors:	Wenjia Ren (16469833) (author), Linjun Liu (16469830) (author), Haojia Zhu (20485284) (author), Ke Xu (134879) (author), Yubai Zhou (3931427) (author)
Published:	2024
Subjects:	Biochemistry Medicine Cell Biology Molecular Biology Developmental Biology Cancer Biological Sciences not elsewhere classified uncharacterized scaffolding protein inhibitors partially reversed common histological subtype crucial potential target high mortality rate akt signaling pathway akt pathway activators sh2d5 positively regulates sh2d5 may serve luad remains unclear emt processes induced lung adenocarcinoma cells found aberrant expression potential mechanisms lung cancer high incidence high expression sh2d5 promotes sh2d5 influenced study demonstrated rescue experiments present study poor prognosis phosphorylation levels mesenchymal transition luad tissues luad patients luad cells late stages closely associated
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SH2D5 knockdown inhibits lung adenocarcinoma (LUAD) cell proliferation, migration and invasion.

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