Intraperitoneal infection of STM WT and STM ΔpflB lead to no significant change in the bacterial burden in blood, liver, and spleen 3 dpi.

A. Schematic showing the protocol followed for determining the organ burden of STM WT and STM ΔpflB 3 days post intraperitoneal infection B-D. Bacterial burden of STM WT and STM ΔpflB in blood (B), liver (C), and spleen (D). Data is represented as Mean ...

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Hlavní autor:	Debapriya Mukherjee (9961289) (author)
Další autoři:	Salik Noor (21761072) (author), Tamoghna Mukherjee (22184148) (author), Mahavir Singh (70735) (author), Dipshikha Chakravortty (230557) (author)
Vydáno:	2025
Témata:	Microbiology Cell Biology Molecular Biology Cancer Science Policy Infectious Diseases Environmental Sciences not elsewhere classified Biological Sciences not elsewhere classified Chemical Sciences not elsewhere classified maintaining ph balance div >< p chain fatty acids elevated intracellular ph 1 genes (< intracellular formate modulates intracellular formate pool enhances stm invasion intracellular pool 1 genes intracellular formate use formate formate lyase signalling molecule scfa found salmonella </ reduced flagellation prgh </ pflb </ pathogenicity island invasion switch increased expression hila </ findings underscore encodes pyruvate derived short csrb </ csra /<
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Intraperitoneal infection of STM WT and STM Δ<i>pflB</i> lead to no significant change in the bacterial burden in blood, liver, and spleen 3 dpi.

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