RIN1 contributed to the developmental switch of synaptic GluN2B to GluN2A receptors in spinal cord SOM<sup>+</sup> interneurons.

RIN1 contributed to the developmental switch of synaptic GluN2B to GluN2A receptors in spinal cord SOM<sup>+</sup> interneurons.

<p><b>(A)</b> Representative NMDAR-EPSCs and AMPAR-EPSCs recorded from P7 to P28. <b>(B and C)</b> Comparisons of NMDAR/AMPAR ratios (B) and decay kinetics of NMDAR-EPSCs (C) recorded from male and female mice. <i>*P</i> = 0.035, <i>**P</i> = 0.0...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلف الرئيسي: Hu-Hu Bai (7296326) (author)
مؤلفون آخرون: Qi Zhang (28502) (author), Shu-Jin Wu (22766054) (author), Yu-Bo Gao (22766057) (author), Juan Li (146180) (author), Xue Bai (129175) (author), Xu Yang (112496) (author), Xiao-Xue Liu (4068775) (author), Jia-Ning Dang (22766060) (author), Xian Yang (584213) (author), Zhan-Wei Suo (7296338) (author), Xiao-Dong Hu (2390053) (author)
منشور في: 2025
الموضوعات:
Cell Biology
Neuroscience
Science Policy
Mental Health
Infectious Diseases
Biological Sciences not elsewhere classified
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sensory experience regulate
rab interactor 1
spinal cord somatostatin
rin1 regulates developmental
rin1 differentially regulated
mediated pathological switch
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rin1 protein
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synaptic trafficking
synaptic plasticity
synaptic n
synapse maturation
subunit stoichiometry
spinal som
specific protein
significant increase
related plasticity
protein level
progressively increased
nmdars ),
new round
neuronal activity
neurological disorders
nerve injury
molecular mechanisms
mature synapses
low early
key component
important role
data showed
data revealed
critical determinant
containing nmdars
analgesic efficacy
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