Inactivation of the identified genes causes improper chromosome segregation.

Inactivation of the identified genes causes improper chromosome segregation.

<p>(<i>A</i>) P<i>xol-1</i>::GFP induction in animals with indicated RNAi treatments. (<i>B</i>) Penetrance of embryonic lethality in animals with indicated RNAi treatments. <i>n</i> = 90–110 per group.</p> <p>(TIF)</p>

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Bibliographic Details
Main Author:	Xiaomeng Yang (702951) (author)
Other Authors:	Ruichen Wei (21181914) (author), Fanfan Meng (8174745) (author), Dianchen Liu (21181917) (author), Xuan Gong (562445) (author), Gary Ruvkun (59736) (author), Wei Wei (21173) (author)
Published:	2025
Subjects:	Biochemistry Cell Biology Genetics Science Policy Environmental Sciences not elsewhere classified Biological Sciences not elsewhere classified performed rnai screens genome instability lead div >< p specify mitochondrial abundance potentially protective role surveilling mitochondrial fission regulate mitochondrial fission mitochondrial fission surveillance another 11 genes 300 random genes mitigate dysregulation caused caenorhabditis elegans </ chromosome segregation integrity mitochondrial fission elegans </ chromosome segregation mitochondrial dynamics potential role chromosome missegregation uneven activation tightly regulated screening paradigm scale screen regulatory pathways possibly independent physiological states less explored kinase library cell division
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