Supplemental table 1.

<div><p>Background</p><p>Ferroptosis is a nonapoptotic form of cell death characterized by lipid peroxidation and intracellular iron accumulation. OA is a prevalent joint disease, and as OA progresses, inflammation or iron overload can induce ferroptosis in chondrocytes. Howe...

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Бібліографічні деталі
Автор:	Jiexiang Yang (19980594) (author)
Інші автори:	Jing Jiang (130103) (author), Jing Wang (6206297) (author), Lin Luo (429475) (author)
Опубліковано:	2025
Предмети:	Biochemistry Cell Biology Molecular Biology Cancer prevalent joint disease potential key gene play important roles novel therapeutic strategy medial meniscus ligament experimental results showed cell death characterized associated geo datasets mediated p53 acetylation inhibits p53 acetylation enhancing p53 acetylation intracellular iron accumulation thereby increasing ferroptosis suppressing trim33 expression oa mouse model related gene sat1 xlink "> ferroptosis axis promotes ferroptosis p53 axis xlink "> sat1 promotes related indicators iron overload induce ferroptosis western blot vivo </ vitro </ regulating trim33 oa progresses oa pathogenesis nonapoptotic form lipid peroxidation downregulating trim33 data indicate close association biological functions atdc5 cells analyzing oa
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Supplemental table 1.

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