BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).

(A and B) BACH1 protein level was detected in mouse (A) and human (B) melanoma cells under the treatment of either DMSO, Nocodazole (NOC), or NOC plus Volasertib. (C and D) Immunoblots of BACH1 protein level in the mouse (C) and human (D) melanoma cells...

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में बचाया:

ग्रंथसूची विवरण
मुख्य लेखक:	Fengyi Mao (15073242) (author)
अन्य लेखक:	Sai Wu (6096290) (author), Derek B. Allison (15141675) (author), Daheng He (9189701) (author), Yifan Kong (4458616) (author), Chaohao Li (15141681) (author), Zhiguo Li (431917) (author), Yanquan Zhang (14930570) (author), Xinyi Wang (559030) (author), Qiongsi Zhang (2818111) (author), Chi Wang (131921) (author), Xiaoqi Liu (45630) (author)
प्रकाशित:	2025
विषय:	Cell Biology Genetics Molecular Biology Developmental Biology Cancer Hematology Biological Sciences not elsewhere classified suppress tumor growth like kinase 1 crucial transcription factor cnc homolog 1 clinical database analysis shortened mice survival >< sup >< div >< p cutaneous melanoma based v600e </ sup patient survival pten </ ca </ ></ sup synergistic efficiency novel function negatively correlates genes involved cell cycle cancer progression cancer metabolism bach1 ), >- deficiency
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BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).

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