BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).

(A and B) BACH1 protein level was detected in mouse (A) and human (B) melanoma cells under the treatment of either DMSO, Nocodazole (NOC), or NOC plus Volasertib. (C and D) Immunoblots of BACH1 protein level in the mouse (C) and human (D) melanoma cells...

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Bibliografiset tiedot
Päätekijä:	Fengyi Mao (15073242) (author)
Muut tekijät:	Sai Wu (6096290) (author), Derek B. Allison (15141675) (author), Daheng He (9189701) (author), Yifan Kong (4458616) (author), Chaohao Li (15141681) (author), Zhiguo Li (431917) (author), Yanquan Zhang (14930570) (author), Xinyi Wang (559030) (author), Qiongsi Zhang (2818111) (author), Chi Wang (131921) (author), Xiaoqi Liu (45630) (author)
Julkaistu:	2025
Aiheet:	Cell Biology Genetics Molecular Biology Developmental Biology Cancer Hematology Biological Sciences not elsewhere classified suppress tumor growth like kinase 1 crucial transcription factor cnc homolog 1 clinical database analysis shortened mice survival >< sup >< div >< p cutaneous melanoma based v600e </ sup patient survival pten </ ca </ ></ sup synergistic efficiency novel function negatively correlates genes involved cell cycle cancer progression cancer metabolism bach1 ), >- deficiency
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BTB domain and CNC homolog 1 (BACH1) is stabilized by Polo-like kinase 1 (PLK1).

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