Epac activation increased RLC phosphorylation via PLC/PKC.

<p>(A) Representative SDS-PAGE patterns of skinned myocardium prepared from WT and treated with 8CPT-AM (5 μM for 30 min) in the presence or absence of PLC inhibitor (U73122, 5 μM), PKC inhibitor (BIM, 1 μM), CaMKII inhibitor (KN-93, 2 μM), MLCK inhibitor (ML-7, 10 μM) or ZIPK inhibitor (HS38,...

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Main Author:	Yoshiki Ohnuki (576298) (author)
Other Authors:	Kenji Suita (750993) (author), Misao Ishikawa (6589574) (author), Yasumasa Mototani (750991) (author), Megumi Nariyama (6589580) (author), Aiko Ito (6589571) (author), Ichiro Matsuo (249122) (author), Yoshio Hayakawa (8043413) (author), Akinaka Morii (12709672) (author), Takao Mitsubayashi (20995465) (author), Yasutake Saeki (576297) (author), Yoshihiro Ishikawa (324232) (author), Satoshi Okumura (324225) (author)
Published:	2025
Subjects:	Biochemistry Cell Biology Genetics Molecular Biology Physiology Developmental Biology myosin binding protein myofilament proteins troponin induces protein kinase exchange protein activated 60 &# 8201 increased cardiac output 1 week ), selective camp analogue independent camp effector wt mice treated mice treated camp ), epac1 ), control mice cardiac myofilaments cardiac function tension cost skinned myocardium significantly greater significantly decreased results suggest much greater living myocardium isoproterenol infusion generating capacity energetic cost cyclic amp calcium sensitivity atpase activity also examined
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Epac activation increased RLC phosphorylation via PLC/PKC.

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