SRPIN340 treatment does not enhance viral genome integration.

<p>The cells used for the other experiments in the manuscript (<a href="http://www.plospathogens.org/article/info:doi/10.1371/journal.ppat.1012697#ppat.1012697.s001" target="_blank">S1 Fig</a>) were unavailable for this experiment. The monolayer-cultured and dif...

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Bibliographic Details
Main Author:	Arwa A. A. Faizo (21030140) (author)
Other Authors:	Clare Bellward (21030143) (author), Hegel R. Hernandez-Lopez (21030146) (author), Andrew Stevenson (9143975) (author), Quan Gu (204269) (author), Sheila V. Graham (11864634) (author)
Published:	2025
Subjects:	Biochemistry Cell Biology Genetics Molecular Biology Immunology Developmental Biology Cancer Infectious Diseases Virology Biological Sciences not elsewhere classified pathway analysis showed novel antiviral therapies cause precancerous lesions cause benign lesions called &# 8220 positively regulate splicing phosphorylate sr proteins l1 late proteins sr protein kinases sr protein activity srpin340 treatment led since srpk1 controls hpv life cycle splicing factor kinase e2 viral replication viral replication protein family alternative splicing transcription factor late events viral mrnas inhibit replication three days therapeutic strategy srpin340 inhibits specific inhibitor sindbis virus rna processing nuclear entry may progress malignant tumours infect epithelia hpvs infecting hpv16 infection hpv e4 full suite epithelial barrier dna repair chromatin conformation cellular location arginine rich arginine domains anogenital region also observed 935 rnas
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SRPIN340 treatment does not enhance viral genome integration.

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