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<div><p>All cells possess mechanisms to maintain and replicate their genomes, whose integrity and transmission are constantly challenged by DNA damage and replication impediments. In eukaryotes, the protein kinase Ataxia-Telangiectasia and Rad3-related (ATR), a member of the phosphatidyl...

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Bibliografski detalji
Glavni autor:	Gabriel L. A. da Silva (22676649) (author)
Daljnji autori:	Jeziel D. Damasceno (9073663) (author), Jennifer A. Black (14411790) (author), Craig Lapsley (6082646) (author), Richard McCulloch (12918) (author), Luiz R. O. Tosi (9073666) (author)
Izdano:	2025
Teme:	Biophysics Biochemistry Microbiology Cell Biology Genetics Molecular Biology Developmental Biology Science Policy Infectious Diseases Biological Sciences not elsewhere classified Physical Sciences not elsewhere classified major &# 8217 div >< p data reveals loss cells possess mechanisms remarkably plastic genome cas9 genome editing like family acts protein kinase ataxia atr depends upon delayed dna replication stranded dna accumulation leishmania major </ dna damage kinase dna damage genome stability leishmania </ dna metabolism dna injuries atr acts replication stress replication impediments work shows whose integrity terminal knockout smaller chromosomes protozoan parasite phosphatidylinositol 3 nuclear localisation mycatrδc -/-). mycatr ), master regulator larger chromosomes key role increased levels functional relevance eukaryotic response deletion results constantly challenged atr plays atr homolog atr ),
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