TGFBR1 gene silencing inhibits cardiac hypertrophy.

(A) Representative western blot images of TGFBR1, p-MEK1/2, p-ERK1/2, p-p38, and p-JNK1/2 in the left-ventricular samples from each group of mice; n = 4 mice per group. (B) Quantification of the protein expression of TGFBR1, p-MEK1/2, p-ERK1/2, p-p38, a...

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محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Xuanyang Shen (22145910) (author)
مؤلفون آخرون:	Weidong Li (292436) (author), Xiaolu Jiang (627400) (author), Hongfu Wen (22145913) (author), Yuan Shen (194742) (author), Wen Zhang (7555) (author), Yongjian Peng (22145916) (author), Guoping Kang (22145919) (author)
منشور في:	2025
الموضوعات:	Biochemistry Medicine Cell Biology Genetics Molecular Biology Physiology Immunology Developmental Biology Cancer Mental Health Infectious Diseases Biological Sciences not elsewhere classified Chemical Sciences not elsewhere classified preserved ejection fraction potential therapeutic target myocardial tgfbr1 expression myocardial collagen synthesis ameliorating myocardial remodeling activated protein kinase reducing myocardial hypertrophy attenuating cardiac fibrosis hfpef remains uncertain 3 signaling pathway signaling pathway cardiac hypertrophy profibrotic impact prior research principal factor mediated panoptosis mechanistic level hfpef ). heart failure findings indicate
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TGFBR1 gene silencing inhibits cardiac hypertrophy.

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