ISG15 expression and secretion is induced in human primary cells and mouse female genital tract organoids upon Ct infection.

<p>a. ISG15 expression in human cells infected with Ct. HUVEC, Fimb, EcCxEp, and HeLa 229 cells were either left uninfected, infected with Ct (MOI 1), or stimulated with human IFN-α (100 ng/ml) for 36h. ISG15, HSP60 (chlamydial infection), and GAPDH (loading control) expression in cell lysates...

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التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Yongxia Guo (8564418) (author)
مؤلفون آخرون:	Sigrun V. Stulz (21681424) (author), David Komla Kessie (11870621) (author), Nadine Vollmuth (15301711) (author), Tommaso Torcellan (21681427) (author), Klaus-Peter Knobeloch (50254) (author), Georg Gasteiger (2314) (author), Thomas Rudel (127432) (author)
منشور في:	2025
الموضوعات:	Microbiology Cell Biology Genetics Ecology Immunology Infectious Diseases uterine horn pathology immunoregulatory cytokine essential div >< p critical dual function 18ra ), demonstrating controlling bacterial load murine infection model controlling chlamydial infection human primary cells chlamydia trachomatis </ 18 receptor alpha nk cells lacking secreted isg15 induced chlamydia </ bacterial infections nk cells unconjugated form ubiquitin family stimulatory effect stimulated gene process known polarizing signal natural killer intracellular pathogens inflammatory responses infected isg15 host resistance host proteins exist either covalently attached completely abolished cellular immunity 10 levels
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ISG15 expression and secretion is induced in human primary cells and mouse female genital tract organoids upon Ct infection.

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