iPSC-derived HCOs are permissive to EBOV and MARV infection.

<p>Human iPSC-derived colonic organoids (HCOs) were infected on day 35 of differentiation with EBOV or MARV at an MOI of 10. (A–C, EBOV infection; E–G, MARV infection) Viral infection was evaluated at 1 and 3 dpi using confocal microscopy. Immunofluorescence staining was performed with antibod...

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Bibliografski detalji
Glavni autor:	Elizabeth Y. Flores (22676699) (author)
Daljnji autori:	Adam J. Hume (6107039) (author), Judith Olejnik (367849) (author), Aditya Mithal (22676702) (author), Andrew D’Amico (2248762) (author), MengWei Yang (22676705) (author), Pushpinder Bawa (733756) (author), Feiya Wang (21075016) (author), Aoife K. O’Connell (15462382) (author), Anna Tseng (20843165) (author), Nicholas A. Crossland (5500244) (author), Gustavo Mostoslavsky (110612) (author), Elke Mühlberger (45516) (author)
Izdano:	2025
Teme:	Biochemistry Microbiology Cell Biology Genetics Molecular Biology Physiology Biotechnology Immunology Cancer Mental Health Infectious Diseases Plant Biology Virology Biological Sciences not elsewhere classified Chemical Sciences not elsewhere classified xlink "> gastrointestinal support viral replication providing mechanistic insight induced secretory diarrhea colonic epithelial responses model ebola virus mediated ion transport human gi pathology ion transport severe diarrhea human ipsc stimulated genes specific induction powerful system pathogen interactions marburg virus junctional disruption including apical filovirus disease filoviral disease dissect host delayed virus colonic organoids central contributor
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iPSC-derived HCOs are permissive to EBOV and MARV infection.

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