Infection of CD133+ humanized mice resulted in decrease in pathogenicity in HTLV-1-CTCF infected mice.

<p>Comparison of A) lymphocyte and B) neutrophil percentages in the peripheral blood in HTLV-1-WT, -p12stop and -CTCF infected mice at the time of necropsy. C) Blood smear of HTLV-1-WT infected mouse #5 at 8 weeks of infection with wbc count of 142,000/µl in which flower cells are evident. * i...

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محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Ancy Joseph (5634794) (author)
مؤلفون آخرون:	Xiaogang Cheng (8166234) (author), John Harding (3445103) (author), Jacob Al-Saleem (659199) (author), Patrick Green (719193) (author), Malachi Griffith (66161) (author), Deborah Veis (21474978) (author), Daniel A. Rauch (9317727) (author), Lee Ratner (73067) (author)
منشور في:	2025
الموضوعات:	Medicine Microbiology Cell Biology Molecular Biology Immunology Cancer Science Policy Mental Health Hematology Infectious Diseases Virology Biological Sciences not elsewhere classified host cell genome abolishes ctcf binding ctcf cell line ctcf virus compared three cell lines div >< p ctcf binding site infected mice suggests cell leukemia virus ctcf infected mice 1 provirus genome p12stop infected mice binding site cell lines cell leukemia mice infected cell counts virus integrates wt virus vivo </ viral expression transcriptionally active stop codon significantly lower scrnaseq analysis proviral load previous studies peripheral blood mouse model initiate infection inactive regions histone methylation findings indicate direct correlation dependent manner chromatin structure 1 infection
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Infection of CD133+ humanized mice resulted in decrease in pathogenicity in HTLV-1-CTCF infected mice.

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