Overexpression of SH2D5 promotes lung adenocarcinoma (LUAD) cell proliferation, migration and invasion.

<p>(A, B) Transfection efficiencies after overexpression of SH2D5 in HCC827 and A549 cells determined by western blot. (C-E) The proliferation ability of vector cells and oeSH2D5 cells in HCC827 and A549 was detected by CCK8 and colony formation assays. (F-I) The wound healing and transwell as...

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Bibliographic Details
Main Author:	Licheng Du (16469836) (author)
Other Authors:	Wenjia Ren (16469833) (author), Linjun Liu (16469830) (author), Haojia Zhu (20485284) (author), Ke Xu (134879) (author), Yubai Zhou (3931427) (author)
Published:	2024
Subjects:	Biochemistry Medicine Cell Biology Molecular Biology Developmental Biology Cancer Biological Sciences not elsewhere classified uncharacterized scaffolding protein inhibitors partially reversed common histological subtype crucial potential target high mortality rate akt signaling pathway akt pathway activators sh2d5 positively regulates sh2d5 may serve luad remains unclear emt processes induced lung adenocarcinoma cells found aberrant expression potential mechanisms lung cancer high incidence high expression sh2d5 promotes sh2d5 influenced study demonstrated rescue experiments present study poor prognosis phosphorylation levels mesenchymal transition luad tissues luad patients luad cells late stages closely associated
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Overexpression of SH2D5 promotes lung adenocarcinoma (LUAD) cell proliferation, migration and invasion.

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