ISG15 induces polarization of wild-type and ISG15−/− macrophages.

a. Schematic illustration of the differentiation and subtypes of BMDMs. Bone marrow cells were isolated from wildtype and ISG15−/− mice and cultured with M-CSF stimulation for 7 days, during which hematopoietic stem cells (HSCs) developed into macrophages (BMDMs). BMD...

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Bibliographic Details
Main Author:	Yongxia Guo (8564418) (author)
Other Authors:	Sigrun V. Stulz (21681424) (author), David Komla Kessie (11870621) (author), Nadine Vollmuth (15301711) (author), Tommaso Torcellan (21681427) (author), Klaus-Peter Knobeloch (50254) (author), Georg Gasteiger (2314) (author), Thomas Rudel (127432) (author)
Published:	2025
Subjects:	Microbiology Cell Biology Genetics Ecology Immunology Infectious Diseases uterine horn pathology immunoregulatory cytokine essential div >< p critical dual function 18ra ), demonstrating controlling bacterial load murine infection model controlling chlamydial infection human primary cells chlamydia trachomatis </ 18 receptor alpha nk cells lacking secreted isg15 induced chlamydia </ bacterial infections nk cells unconjugated form ubiquitin family stimulatory effect stimulated gene process known polarizing signal natural killer intracellular pathogens inflammatory responses infected isg15 host resistance host proteins exist either covalently attached completely abolished cellular immunity 10 levels
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ISG15 induces polarization of wild-type and ISG15<sup>−/−</sup> macrophages.

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