The expression of granzyme B in infiltrated CD8<sup>+</sup> T cells in the brain of <i>Il17rc</i><sup><i>-/-</i></sup> mice.

The expression of granzyme B in infiltrated CD8<sup>+</sup> T cells in the brain of <i>Il17rc</i><sup><i>-/-</i></sup> mice.

<p>Eight to nine weeks old <i>Il17rc</i><sup><i>-/-</i></sup> (n = 4) and WT mice (n = 4) were infected with 20 PFU of WNV via f.p. inoculation, and brain tissue was collected on D8 p.i. The brain leukocytes were characterized by flow cytometry after probing...

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Autore principale:	Farzana Nazneen (9644837) (author)
Altri autori:	Biswas Neupane (2167513) (author), Yao Chen (149006) (author), Shazeed-Ul Karim (21688918) (author), Zongbing You (41988) (author), Weiguo Cui (3390203) (author), Fengwei Bai (139564) (author)
Pubblicazione:	2025
Soggetti:	Biochemistry Medicine Microbiology Cell Biology Genetics Molecular Biology Neuroscience Immunology Cancer Mental Health Infectious Diseases Virology Biological Sciences not elsewhere classified west nile virus stimulating immune responses higher energy demand div >< p borne neurotropic flavivirus >< sup >< sup >+</ sup higher viral load ex vivo </ specific therapeutic intervention 17rc deficient (< 17ra deficient (< 17 receptor c mtor signaling axis 17a signaling activated ></ sup viral encephalitis 17 receptor signaling promotes signaling pathway signaling induced il17rc </ il17ra </ united states public health previous studies parasite clearance obligatory co molecular mechanism mediated metabolism mammalian target major cause findings reveal expression levels effector functions counterpart controls continuous threat conditional knockout
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