The initial infection of alphaherpesvirus is delayed by Arc knockdown.

Neuronally differentiated SH-SY5Y cells were treated with siRNA #1 (#1) or siRNA #2 (#2) for Arc knockdown. Negative target siRNA-treated cells served as controls (Ctrl). PRV- (A) or HSV-1-infected (B) cells at an MOI of 10. The amount of PRV or HSV-1 was determined by we...

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التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Hiroko Kobayashi (794285) (author)
مؤلفون آخرون:	Mitsuki Yasukochi (20422323) (author), Masayuki Horie (114530) (author), Yasuko Orba (251924) (author), Hirofumi Sawa (251933) (author), Kan Fujino (114526) (author), Satoshi Taharaguchi (12190780) (author)
منشور في:	2024
الموضوعات:	Biochemistry Medicine Neuroscience Physiology Immunology Cancer Mental Health Infectious Diseases Virology Biological Sciences not elsewhere classified various host factors transcriptional regulatory genes stranded dna viruses providing fundamental insights major capsid protein host neuronal cells detailed mechanisms underlying derived domesticated genes derived domesticated gene ensure efficient infection abnormal expression leads derived protein arc derived arc interactions viral life cycle div >< p enhanced arc expression alphaherpesvirus infection upregulate arc arc interacts arc </ viral lifecycle viral factor viral attachment vitro </ pseudorabies virus provide evidence physiological significance neurotropic double neurological disorders mass spectrometry important role identified icp0 early stages
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The initial infection of alphaherpesvirus is delayed by <i>Arc</i> knockdown.

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