Secreted ISG15 modulates the immune response during Ct FGT infection.

<p>Schematic representation of the immunomodulatory functions of ISG15 in the FGT during Ct infection. After Ct infection, genital epithelial cells release ISG15. ISG15, in synergy with IL-12 and IL-18, potentiates the production of IFN-γ by NK cells, contributing to the immune defense against...

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محفوظ في:

التفاصيل البيبلوغرافية
المؤلف الرئيسي:	Yongxia Guo (8564418) (author)
مؤلفون آخرون:	Sigrun V. Stulz (21681424) (author), David Komla Kessie (11870621) (author), Nadine Vollmuth (15301711) (author), Tommaso Torcellan (21681427) (author), Klaus-Peter Knobeloch (50254) (author), Georg Gasteiger (2314) (author), Thomas Rudel (127432) (author)
منشور في:	2025
الموضوعات:	Microbiology Cell Biology Genetics Ecology Immunology Infectious Diseases uterine horn pathology immunoregulatory cytokine essential div >< p critical dual function 18ra ), demonstrating controlling bacterial load murine infection model controlling chlamydial infection human primary cells chlamydia trachomatis </ 18 receptor alpha nk cells lacking secreted isg15 induced chlamydia </ bacterial infections nk cells unconjugated form ubiquitin family stimulatory effect stimulated gene process known polarizing signal natural killer intracellular pathogens inflammatory responses infected isg15 host resistance host proteins exist either covalently attached completely abolished cellular immunity 10 levels
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Secreted ISG15 modulates the immune response during Ct FGT infection.

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