Figure 6 from Bcl-xL Is a Key Mediator of Apoptosis Following KRASG12C Inhibition in KRASG12C-mutant Colorectal Cancer

AZ’1569-acquired resistant cells exhibit increased PD-L1 expression and a proinflammatory phenotype. A, Left: RW7213 parental and AZ’1569-resistant clones (No. 2, No. 3, and No. 4) were treated for 72 hours with indicated concentrations of AZ’1569 or sotorasib, and cell v...

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Auteur principal:	Hajrah Khawaja (15090810) (author)
Autres auteurs:	Rebecca Briggs (15090813) (author), Cheryl H. Latimer (15090816) (author), Mustasin Rassel (15090819) (author), Daryl Griffin (15090822) (author), Lyndsey Hanson (15090825) (author), Alberto Bardelli (15046646) (author), Frederica Di Nicolantonio (15090828) (author), Simon S. McDade (14939679) (author), Christopher J. Scott (15090831) (author), Shauna Lambe (15090834) (author), Manisha Maurya (15090837) (author), Andreas U. Lindner (15033719) (author), Jochen H.M. Prehn (15033731) (author), Jose Sousa (15090840) (author), Chris Winnington (15090843) (author), Melissa J. LaBonte (15090846) (author), Sarah Ross (15090849) (author), Sandra Van Schaeybroeck (15090852) (author)
Publié:	2025
Sujets:	Cancer Cancer Biology Therapeutic Research and Development Drug Resistance Novel mechanisms Gastrointestinal Cancers Colorectal cancer Oncogenes & Tumor Suppressors Kras
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Figure 6 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer

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