Canonical histones do not stoichiometrically localize to nuclear infecting HSV-1 genomes.

<p><b>(A)</b> HFt cells were infected with WT HSV-1 (MOI of 3 PFU/cell). Chromatin extracts were prepared at 90 mins post-infection (mpi; post-addition of virus) and subjected to ChIP using ChIP-grade anti-histone H2B or histone H3 antibodies and species-matched IgG (negative contr...

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Bibliographic Details
Main Author:	Ashley P. E. Roberts (4744257) (author)
Other Authors:	Anne Orr (205891) (author), Victor Iliev (6499388) (author), Lauren Orr (11798082) (author), Steven McFarlane (205895) (author), Zhousiyu Yang (22097351) (author), Ilaria Epifano (11864619) (author), Colin Loney (418948) (author), Mila Collados Rodriguez (22097354) (author), Anna R. Cliffe (12982392) (author), Kristen L. Conn (17234788) (author), Chris Boutell (205885) (author)
Published:	2025
Subjects:	Medicine Microbiology Genetics Molecular Biology Cancer Infectious Diseases Virology Environmental Sciences not elsewhere classified Biological Sciences not elsewhere classified xlink "> herpesviruses upon nuclear entry first 90 minutes resolution quantitative imaging independent histone h3 chromatin modifying enzymes h4 chaperone daxx variant histone h3 1 genome decompaction 1 lytic infection genome resolution viral chromatin genome expansion chromatin immunoprecipitation variant histones wide range viral dna use high ubiquitous pathogens transcriptional regulation spatial proximity spatial localization key role infected cells host response host factors genomes within genomes associate findings pertinent extensively studied disperse daxx data support bulk populations actively recruited
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Canonical histones do not stoichiometrically localize to nuclear infecting HSV-1 genomes.

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